Directorio de personas
Josep Enric Esquerda Colell

Josep Enric Esquerda Colell

Grado: Doctor/a

973 702 427
josep.esquerda(ELIMINAR)@udl.cat

ResearcherID: http://www.researcherid.com/rid/A-7377-2010

Grupos

Publicaciones

  • Oppenheim, RW; CALDERO, J; Cuitat, D; Esquerda, J; Ayala, V; Prevette, D; Wang, SW

    Rescue of developing spinal motoneurons from programmed cell death by the GABA(A) agonist muscimol acts by blockade of neuromuscular activity and increased intramuscular nerve branching.

    MOLECULAR AND CELLULAR NEUROSCIENCE 22 331-343. .

    [doi:10.1016/S1044-7431(02)00020-9]

  • Ribera, J; Ayala, V; ESQUERDA, JE

    c-Jun-like immunoreactivity in apoptosis is the result of a crossreaction with neoantigenic sites exposed by caspase-3-mediated proteolysis.

    Journal Of Histochemistry & Cytochemistry 50 961-972. .

    [doi:10.1177/002215540205000710]

  • Winseck, AK; CALDERO, J; CIUTAT, D; Prevette, D; Scott, SA; Wang, GY; ESQUERDA, JE; Oppenheim, RW

    In vivo analysis of Schwann cell programmed cell death in the embryonic chick: regulation by axons and glial growth factor.

    JOURNAL OF NEUROSCIENCE 22 4509-4521. .

    [doi:10.1523/JNEUROSCI.22-11-04509.2002]

  • Serrando, M; CASANOVAS, A; ESQUERDA, JE

    Occurrence of glutamate receptor subunit 1-containing aggresome-like structures during normal development of rat spinal cord interneurons.

    Journal Of Comparative Neurology 442 23-34. .

    [doi:10.1002/cne.1419]

  • Tarabal, O; CALDERO, J; Llado, J; Oppenheim, RW; ESQUERDA, JE

    Long-lasting aberrant tubulovesicular membrane inclusions accumulate in developing motoneurons after a sublethal excitotoxic insult: a possible model for neuronal pathology in neurodegenerative disease.

    JOURNAL OF NEUROSCIENCE 21 8072-8081. .

  • Casas, C; Ribera, J; ESQUERDA, JE

    Antibodies against c-Jun N-terminal peptide cross-react with neo-epitopes emerging after caspase-mediated proteolysis during apoptosis.

    JOURNAL OF NEUROCHEMISTRY 77 904-915. .

    [doi:10.1046/j.1471-4159.2001.00314.x]

  • CASANOVAS, A; Ribera, J; Hager, G; Kreutzberg, GW; ESQUERDA, JE

    c-Jun regulation in rat neonatal motoneurons postaxotomy.

    Journal Of Neuroscience Research 63 469-479. .

    [doi:10.1002/jnr.1041]

  • CASANOVAS, A; Olmos, G; Ribera, J; Boronat, MA; ESQUERDA, JE; Garcia-Sevilla, JA

    Induction of reactive astrocytosis and prevention of motoneuron cell death by the I(2)-imidazoline receptor ligand LSL 60101.

    BRITISH JOURNAL OF PHARMACOLOGY 130 1767-1776. .

    [doi:10.1038/sj.bjp.0703485]

  • Tarabal, O; LladoJ, OJ; CALDERO, J; Ribera, J; CIUTAT, D; CASANOVAS, A; Ayala, V; Serrando, M; Casas, C; Sarasa, M; ESQUERDA, JE

    Chronic NMDa treatment results in a prevention of acute excitotoxic injury at tm chick embryo motoneurons and induces permanent changes in intracellular membrane compartimentation.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 38-38. .

  • CALDERO, J; Llado, J; Ribera, J; Tarabal, O; CIUTAT, D; CASANOVAS, A; Ayala, V; Casas, C; Serrando, M; Oppenheim, RW; ESQUERDA, JE

    Chronic administration of n-methyl-d-aspartate (NMDa) prevents excitotoxicity and normal and induced programmed cell death in spinal cord motoneurons.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 394-394. .

  • CASANOVAS, A; Ribera, J; Hager, G; Kreutzberg, GW; Casas, C; Ayala, V; CALDERO, J; CIUTAT, D; Tarabal, O; Llado, J; Serrando, M; ESQUERDA, JE

    Differential expression of c-jun after axotomy in neonatals suggest different functions in programmed cell death and axonal regeneration.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 8-8. .

  • Casas, C; Ayala, V; Ribera, J; CALDERO, J; CASANOVAS, A; CIUTAT, D; Tarabal, O; Llado, J; Serrando, M; ESQUERDA, JE

    c-jun-like immunoreactivity specifically associated with apoptosis reveals new epitopes emerged by caspase-3 cleavage.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 8-8. .

Proyectos

  • Preclinical Analisis of new combinatorial treatments for spinal muscular atrophy (SMA): Effects on motorneuron survival, synaptic integrity, and skeletal muscle preservation.
  • El sistema NRG1-ErbBs en la arquitectura molecular de las sinapsis aferentes de tipo C y su significado en las enfermedades de la motoneurona
  • Los aferentes sinápticos de tipo C en las motoneuronas: implicaciones en la fisiopatología y terapia de la esclerosis lateral amiotrófica y de la atrofia muscular espinal (Motoneurona)
  • La esclerosis lateral amiotrófica como enfermedad proteinopática y neuroimflamatoria: estrategias de inmunoterapia (Vacunas y ELA)
  • Excitotoxicidad, plegamiento anómalo de proteinas y mecanismos inmunológicos en la esclerosis lateral amiotrófica
  • Mecanismes excitotòxics, resposta neuroinflamatòria i factors sèrics en l´esclerosi lateral amiotròfica humana i experimental