Directorio de personas
Josep Enric Esquerda Colell

Josep Enric Esquerda Colell

Grado: Doctor/a

973 702 427
josep.esquerda(ELIMINAR)@udl.cat

ResearcherID: http://www.researcherid.com/rid/A-7377-2010

Grupos

Publicaciones

  • Ayala, V; Casas, C; Ribera, J; CALDERO, J; Oppenheim, RW; ESQUERDA, JE

    Specific association of c-Jun-like immunoreactivity but not c-Jun p39 with normal and induced programmed cell death in the chick embryo.

    Journal Of Neurobiology 38 171-190. .

    [doi:10.1002/(SICI)1097-4695(19990205)38:2<171::AID-NEU2>3.0.CO;2-#]

  • Ribera, J; Marsal, J; CASANOVAS, A; Hukkanen, M; Tarabal, O; ESQUERDA, JE

    Nitric oxide synthase in rat neuromuscular junctions and in nerve terminals of Torpedo electric organ: its role as regulator of acetylcholine release.

    Journal Of Neuroscience Research 51 90-102. .

    [doi:10.1002/(SICI)1097-4547(19980101)51:1<90::AID-JNR10>3.0.CO;2-C]

  • CALDERO, J; CIUTAT, D; Llado, J; Castan, E; Oppenheim, RW; ESQUERDA, JE

    Effects of excitatory amino acids on neuromuscular development in the chick embryo.

    Journal Of Comparative Neurology 387 73-95. .

    [doi:10.1002/(SICI)1096-9861(19971013)387:1<73::AID-CNE7>3.0.CO;2-K]

  • Tarabal, O; CALDERO, J; ESQUERDA, JE

    Intramuscular nerve sprouting induced by CNTF is associated with increases in CGRP content in mouse motor nerve terminals.

    NEUROSCIENCE LETTERS 219 60-64. .

    [doi:10.1016/S0304-3940(96)13174-8]

  • CIUTAT, D; CALDERO, J; Oppenheim, RW; ESQUERDA, JE

    Schwann cell apoptosis during normal development and after axonal degeneration induced by neurotoxins in the chick embryo.

    JOURNAL OF NEUROSCIENCE 16 3979-3990. .

  • Tarabal, O; CALDERO, J; Ribera, J; SORRIBAS, A; Lopez, R; Molgo, J; ESQUERDA, JE

    Regulation of motoneuronal calcitonin gene-related peptide (CGRP) during axonal growth and neuromuscular synaptic plasticity induced by botulinum toxin in rats.

    EUROPEAN JOURNAL OF NEUROSCIENCE 8 829-836. .

    [doi:10.1111/j.1460-9568.1996.tb01269.x]

  • Iglesias, M; Soler, RM; Ribera, J; ESQUERDA, JE; Comella, JX

    The carbohydrate N-acetylglucosamine is involved in the guidance of neurites from chick ciliary ganglion neurons through the extracellular matrix of rat skeletal muscle fiber.

    NEUROSCIENCE LETTERS 207 81-84. .

    [doi:10.1016/0304-3940(96)12506-4]

  • CASANOVAS, A; Ribera, J; Hukkanen, M; RiverosMoreno, V; ESQUERDA, JE

    Prevention by lamotrigine, MK-801 and N-omega-nitro-L-arginine methyl ester of motoneuron cell death after neonatal axotomy

    NEUROSCIENCE 71 313-325. .

    [doi:10.1016/0306-4522(95)00461-0]

  • Casanovas A; Ribera J; Hukkanen M; Riveros-Moreno V; Esquerda JE

    Prevention by lamotrigine, MK-801 and N omega-nitro-L-arginine methyl ester of motoneuron cell death after neonatal axotomy.

    NEUROSCIENCE 71 313-325. .

  • Iglesias, M; Soler, RM; Hunter, DD; Ribera, J; ESQUERDA, JE; Comella, JX

    S-laminin and N-acetylgalactosamine located at the synaptic basal lamina of skeletal muscle are involved in synaptic recognition by growing neurites.

    Journal Of Neurocytology 24 903-915. .

    [doi:10.1007/BF01215641]

  • CIUTAT, D; ESQUERDA, JE; CALDERO, J

    EVIDENCE FOR CaLCIUM REGULaTION OF SPINaL-CORD MOTONEURON DEaTH IN THE CHICK-EMBRYO IN-VIVO

    Developmental Brain Research 86 167-179. .

    [doi:10.1016/0165-3806(95)00027-B]

  • COMELLA, JX; SANZRODRIGUEZ, C; ALDEA, M; ESQUERDA, JE

    SKELETaL MUSCLE-DERIVED TROPHIC FaCTORS PREVENT MOTONEURONS FROM ENTERING aN aCTIVE CELL-DEaTH PROGRaM IN-VITRO

    JOURNAL OF NEUROSCIENCE 14 2674-2686. .

Proyectos

  • Preclinical Analisis of new combinatorial treatments for spinal muscular atrophy (SMA): Effects on motorneuron survival, synaptic integrity, and skeletal muscle preservation.
  • El sistema NRG1-ErbBs en la arquitectura molecular de las sinapsis aferentes de tipo C y su significado en las enfermedades de la motoneurona
  • Los aferentes sinápticos de tipo C en las motoneuronas: implicaciones en la fisiopatología y terapia de la esclerosis lateral amiotrófica y de la atrofia muscular espinal (Motoneurona)
  • La esclerosis lateral amiotrófica como enfermedad proteinopática y neuroimflamatoria: estrategias de inmunoterapia (Vacunas y ELA)
  • Excitotoxicidad, plegamiento anómalo de proteinas y mecanismos inmunológicos en la esclerosis lateral amiotrófica
  • Mecanismes excitotòxics, resposta neuroinflamatòria i factors sèrics en l´esclerosi lateral amiotròfica humana i experimental