Anna Maria Casanovas Llorens
Degree: PhD
ana.casanovas(ELIMINAR)@udl.cat
ResearcherID: http://www.researcherid.com/rid/C-3984-2011
Publications
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Casanovas, A; Salvany, S; Lahoz, V; Tarabal, O; Piedrafita, L; Sabater, R; Hernandez, S; Caldero, J; Esquerda, JE
Neuregulin 1-ErbB module in C-bouton synapses on somatic motor neurons: molecular compartmentation and response to peripheral nerve injury
SCIENTIFIC REPORTS 7 40155-40155. .
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SABADO PORCAR, JAVIER; CASANOVAS LLORENS, ANNA MARIA; Rodrigo, H.; Arqué, G.; ESQUERDA COLELL, JOSEP ENRIC
ADVERSE EFFECTS OF A SOD1-PEPTIDE IMMUNOTHERAPY ON SOD1(G93A) MOUSE SLOW MODEL OF AMYOTROPHIC LATERAL SCLEROSIS
NEUROSCIENCE 310 38-50. .
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Puigdellívol-Sánchez A; Giralt A; Casanovas A; Alberch J; Prats-Galino A
Cryostat Slice Irregularities May Introduce Bias in Tissue Thickness Estimation: Relevance for Cell Counting Methods
MICROSCOPY AND MICROANALYSIS 21 893-901. .
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Gallart-Palau X; Tarabal O; Casanovas A; Sábado J; Correa FJ; Hereu M; Piedrafita L; Calderó J; Esquerda JE
Neuregulin-1 is concentrated in the postsynaptic subsurface cistern of C-bouton inputs to alpha-motoneurons and altered during motoneuron diseases
FASEB JOURNAL 28 3618-3632. .
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Sabado, Javier; Casanovas, Anna; Tarabal, Olga; Hereu, Marta; Piedrafita, Ldia; Caldero, Jordi; Esquerda, Josep E.
Accumulation of Misfolded SOD1 in Dorsal Root Ganglion Degenerating Proprioceptive Sensory Neurons of Transgenic Mice with Amyotrophic Lateral Sclerosis
BioMed Research International 2014 -. .
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Sábado J; Casanovas A; HERNÁNDEZ ESTAÑOL, SARA; Piedrafita L; Hereu M; Esquerda JE
Immunodetection of Disease-Associated Conformers of Mutant Cu/Zn Superoxide Dismutase 1 Selectively Expressed in Degenerating Neurons in Amyotrophic Lateral Sclerosis
JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY 72 646-661. .
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Arque, Gloria; CASANOVAS, A.; Dierssen, Mara
Dyrk1A Is Dynamically Expressed on Subsets of Motor Neurons and in the Neuromuscular Junction: Possible Role in Down Syndrome
PLOS ONE 8 -. .
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Texido, Laura; Hernandez, Sara; Martin-Satue, Mireia; Povedano, Monica; Casanovas, Anna; Esquerda, Josep; Marsal, Jordi; Solsona, Caries
Sera from amyotrophic lateral sclerosis patients induce the non-canonical activation of NMDA receptors "in vitro".
NEUROCHEMISTRY INTERNATIONAL 59 954-964. .
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Dachs, Elisabet; Hereu, Marta; Piedrafita, Lidia; Casanovas, Anna; Caldero, Jordi; Esquerda, Josep E.
Defective neuromuscular junction organization and postnatal myogenesis in mice with severe spinal muscular atrophy.
JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY 70 444-461. .
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Hernandez, Sara; Texido, Laura; Caldero, Jordi; Ciutat, Dolors; Piedrafita, Lidia; Casanovas, Anna; Blasi, Joan; Solsona, Carles; Povedano, Monica; Rojas, Ricardo; Illa, Isabel; Caress, James; Prevette, David; Oppenheim, Ronald W.; Milligan, Carol; Esquerda, Josep E.
Increased intramuscular nerve branching and inhibition of programmed cell death of chick embryo motoneurons by immunoglobulins from patients with motoneuron disease.
Journal Of Neuroimmunology 229 157-168. .
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Hernández S; Casanovas A; Piedrafita L; Tarabal O; Esquerda JE
Neurotoxic species of misfolded SOD1G93A recognized by antibodies against the P2X4 subunit of the ATP receptor accumulate in damaged neurons of transgenic animal models of amyotrophic lateral sclerosis.
JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY 69 176-187. .
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Casanovas, Anna; Hernandez, Sara; Tarabal, Olga; Rossello, Jaume; Esquerda, Josep E.
Strong P2X4 purinergic receptor-like immunoreactivity is selectively associated with degenerating neurons in transgenic rodent models of amyotrophic lateral sclerosis.
Journal Of Comparative Neurology 506 75-92. .
Projects
- Preclinical Analisis of new combinatorial treatments for spinal muscular atrophy (SMA): Effects on motorneuron survival, synaptic integrity, and skeletal muscle preservation.
- Los aferentes sinápticos de tipo C en las motoneuronas: implicaciones en la fisiopatología y terapia de la esclerosis lateral amiotrófica y de la atrofia muscular espinal (Motoneurona)
- Mecanismes excitotòxics, resposta neuroinflamatòria i factors sèrics en l´esclerosi lateral amiotròfica humana i experimental