Directori de persones
Elisa Cabiscol Catala

Elisa Cabiscol Catala

Grau: Doctor/a

973 702 281
elisa.cabiscol(ELIMINAR)@udl.cat

ResearcherID: http://www.researcherid.com/rid/A-4584-2009

Publicacions

  • Sanz-Alcázar, A; Portillo-Carrasquer, M; Delaspre, F; Pazos-Gil, M; Tamarit, J; Ros, J; Cabiscol, E

    Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3ß in the impairment of the NRF2 response

    Redox Biology 76 103339-103339. .

    [doi:10.1016/j.redox.2024.103339]

  • Alemany-Perna B; Tamarit J; Cabiscol E; Ros J

    Reply to: "Calcitriol in Friedreich Ataxia".

    MOVEMENT DISORDERS 39 1654-1655. .

    [doi:10.1002/mds.29971]

  • Alemany-Perna, B; Tamarit, J; Cabiscol, E; Delaspre, F; Miguela, A; Huertas-Pons, JM; Quiroga-Varela, A; Ruiz, MM; Dominguez, DL; Torrenta, L; Genis, D; Ros, J

    Calcitriol Treatment Is Safe and Increases Frataxin Levels in Friedreich Ataxia Patients

    MOVEMENT DISORDERS 39 1099-1108. .

    [doi:10.1002/mds.29808]

  • Sanz-Alcázar A; Britti E; Delaspre F; Medina-Carbonero M; Pazos-Gil M; Tamarit J; Ros J; Cabiscol E

    Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.

    Cellular And Molecular Life Sciences 81 12-12. .

    [doi:10.1007/s00018-023-05064-4]

  • Lopez-Mejia IC; Pijuan J; Navaridas R; Santacana M; Gatius S; Velasco A; Castellà G; Panosa A; Cabiscol E; Pinyol M; Coll L; Bonifaci N; Peña LP; Vidal A; Villanueva A; Gari E; Llobet-Navàs D; Fajas L; Matias-Guiu X; Yeramian A

    Oxidative stress-induced FAK activation contributes to uterine serous carcinoma aggressiveness.

    MOLECULAR ONCOLOGY 17 98-118. .

    [doi:10.1002/1878-0261.13346]

  • Medina-Carbonero, M; Sanz-Alcazar, A; Britti, E; Delaspre, F; Cabiscol, E; Ros, J; Tamarit, J

    Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.

    Cellular And Molecular Life Sciences 79 74-74. .

    [doi:10.1007/s00018-021-04100-5]

  • Tamarit J; Britti E; Delaspre F; Medina-Carbonero M; Sanz-Alcázar A; Cabiscol E; Ros J

    Mitochondrial iron and calcium homeostasis in Friedreich ataxia.

    IUBMB LIFE 73 543-553. .

    [doi:10.1002/iub.2457]

  • Rodríguez Colman MJ; Ros J; Cabiscol E

    Mitochondrial Localization of the Yeast Forkhead Factor Hcm1.

    INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 21 -. .

    [doi:10.3390/ijms21249574]

  • Guerrero-Gomez, D; Mora-Lorca, JA; Saenz-Narciso, B; Naranjo-Galindo, FJ; Munoz-Lobato, F; Parrado-Fernandez, C; Goikolea, J; Cedazo-Minguez, A; Link, CD; Neri, C; Sequedo, MD; Vazquez-Manrique, RP; Fernandez-Suarez, E; Goder, V; Pane, R; Cabiscol, E; Askjaer, P; Cabello, J; Miranda-Vizuete, A

    Loss of glutathione redox homeostasis impairs proteostasis by inhibiting autophagy-dependent protein degradation

    CELL DEATH AND DIFFERENTIATION 26 1545-1565. .

    [doi:10.1038/s41418-018-0270-9]

  • Vall-llaura, N; Mir, N; Garrido, L; Vived, C; Cabiscol, E

    Redox control of yeast Sir2 activity is involved in acetic acid resistance and longevity

    Redox Biology 24 101229-101229. .

    [doi:10.1016/j.redox.2019.101229]

  • Gomez-Pastor, R; Burchfiel, ET; Neef, DW; Jaeger, AM; Cabiscol, E; McKinstry, SU; Doss, A; Aballay, A; Lo, DC; Akimov, SS; Ross, CA; Eroglu, C; Thiele, DJ

    Abnormal degradation of the neuronal stress-protective transcription factor HSF1 in Huntington's disease

    Nature Communications 8 14405-14405. .

    [doi:10.1038/ncomms14405]

  • Cabiscol E.; Tamarit J.; Ros J.

    Specificity of Protein Carbonylation and Its Relevance in Aging

    Protein Carbonylation: Principles, Analysis, And Biological Implications 340-383. .

    [doi:10.1002/9781119374947.ch14]

Projectes

  • Alteraciones moleculares en el modelo murino FXNI151F de Ataxia de Friedreich y análisis de nuevas estrategias terapéuticas
  • Uncovering the function of a conserved site in eukaryotic frataxins.
  • PATHOPHYSIOLOGY AND THERAPEUTICS OF FRIENDREICH ATAXIA: MARKERS OR OXIDATIVE STRESS FROM YEAST TO CARDIOMYOCYTES
  • Papel del factor de transcripción Hcm1 en la biogénesis mitocondrial, envejecimiento y la limitación de nutrientes en S. cervisiae
  • Papel funcional del estrés oxidativo y nitrosativo en grandes sistemas biológicos.
  • Estrés oxidativo, envejecimiento celular, homeostasis del hierro y patologías relacionadas en S. cerevisae como modelo celular