Directori de persones
Josep Enric Esquerda Colell

Josep Enric Esquerda Colell

Grau: Doctor/a

973 702 427
josep.esquerda(ELIMINAR)@udl.cat

ResearcherID: http://www.researcherid.com/rid/A-7377-2010

Grups

Publicacions

  • Ribera, J; Ayala, V; ESQUERDA, JE

    c-Jun-like immunoreactivity in apoptosis is the result of a crossreaction with neoantigenic sites exposed by caspase-3-mediated proteolysis.

    Journal Of Histochemistry & Cytochemistry 50 961-972. .

    [doi:10.1177/002215540205000710]

  • Winseck, AK; CALDERO, J; CIUTAT, D; Prevette, D; Scott, SA; Wang, GY; ESQUERDA, JE; Oppenheim, RW

    In vivo analysis of Schwann cell programmed cell death in the embryonic chick: regulation by axons and glial growth factor.

    JOURNAL OF NEUROSCIENCE 22 4509-4521. .

    [doi:20026391]

  • Serrando, M; CASANOVAS, A; ESQUERDA, JE

    Occurrence of glutamate receptor subunit 1-containing aggresome-like structures during normal development of rat spinal cord interneurons.

    Journal Of Comparative Neurology 442 23-34. .

    [doi:10.1002/cne.1419]

  • Tarabal, O; CALDERO, J; Llado, J; Oppenheim, RW; ESQUERDA, JE

    Long-lasting aberrant tubulovesicular membrane inclusions accumulate in developing motoneurons after a sublethal excitotoxic insult: a possible model for neuronal pathology in neurodegenerative disease.

    JOURNAL OF NEUROSCIENCE 21 8072-8081. .

  • Casas, C; Ribera, J; ESQUERDA, JE

    Antibodies against c-Jun N-terminal peptide cross-react with neo-epitopes emerging after caspase-mediated proteolysis during apoptosis.

    JOURNAL OF NEUROCHEMISTRY 77 904-915. .

    [doi:10.1046/j.1471-4159.2001.00314.x]

  • CASANOVAS, A; Ribera, J; Hager, G; Kreutzberg, GW; ESQUERDA, JE

    c-Jun regulation in rat neonatal motoneurons postaxotomy.

    Journal Of Neuroscience Research 63 469-479. .

    [doi:10.1002/jnr.1041]

  • CASANOVAS, A; Olmos, G; Ribera, J; Boronat, MA; ESQUERDA, JE; Garcia-Sevilla, JA

    Induction of reactive astrocytosis and prevention of motoneuron cell death by the I(2)-imidazoline receptor ligand LSL 60101.

    BRITISH JOURNAL OF PHARMACOLOGY 130 1767-1776. .

    [doi:10.1038/sj.bjp.0703485]

  • Tarabal, O; LladoJ, OJ; CALDERO, J; Ribera, J; CIUTAT, D; CASANOVAS, A; Ayala, V; Serrando, M; Casas, C; Sarasa, M; ESQUERDA, JE

    Chronic NMDa treatment results in a prevention of acute excitotoxic injury at tm chick embryo motoneurons and induces permanent changes in intracellular membrane compartimentation.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 38-38. .

  • CALDERO, J; Llado, J; Ribera, J; Tarabal, O; CIUTAT, D; CASANOVAS, A; Ayala, V; Casas, C; Serrando, M; Oppenheim, RW; ESQUERDA, JE

    Chronic administration of n-methyl-d-aspartate (NMDa) prevents excitotoxicity and normal and induced programmed cell death in spinal cord motoneurons.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 394-394. .

  • CASANOVAS, A; Ribera, J; Hager, G; Kreutzberg, GW; Casas, C; Ayala, V; CALDERO, J; CIUTAT, D; Tarabal, O; Llado, J; Serrando, M; ESQUERDA, JE

    Differential expression of c-jun after axotomy in neonatals suggest different functions in programmed cell death and axonal regeneration.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 8-8. .

  • Casas, C; Ayala, V; Ribera, J; CALDERO, J; CASANOVAS, A; CIUTAT, D; Tarabal, O; Llado, J; Serrando, M; ESQUERDA, JE

    c-jun-like immunoreactivity specifically associated with apoptosis reveals new epitopes emerged by caspase-3 cleavage.

    EUROPEAN JOURNAL OF NEUROSCIENCE 12 8-8. .

  • Llado, J; CALDERO, J; Ribera, J; Tarabal, O; Oppenheim, RW; ESQUERDA, JE

    Opposing effects of excitatory amino acids on chick embryo spinal cord motoneurons: excitotoxic degeneration or prevention of programmed cell death.

    JOURNAL OF NEUROSCIENCE 19 10803-10812. .

Projectes

  • Preclinical Analisis of new combinatorial treatments for spinal muscular atrophy (SMA): Effects on motorneuron survival, synaptic integrity, and skeletal muscle preservation.
  • El sistema NRG1-ErbBs en la arquitectura molecular de las sinapsis aferentes de tipo C y su significado en las enfermedades de la motoneurona
  • Los aferentes sinápticos de tipo C en las motoneuronas: implicaciones en la fisiopatología y terapia de la esclerosis lateral amiotrófica y de la atrofia muscular espinal (Motoneurona)
  • La esclerosis lateral amiotrófica como enfermedad proteinopática y neuroimflamatoria: estrategias de inmunoterapia (Vacunas y ELA)
  • Excitotoxicidad, plegamiento anómalo de proteinas y mecanismos inmunológicos en la esclerosis lateral amiotrófica
  • Mecanismes excitotòxics, resposta neuroinflamatòria i factors sèrics en l´esclerosi lateral amiotròfica humana i experimental